Ohio Drivers Eye Exam Chart

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Do I need to file a police report if my license was lost or stolen?
Drivers must report a lost drivers license or stolen drivers license with the NJ police department. This is very important, as it will protect you from possible fraudulent activity and identity theft. You must also report your missing license to drive with the NJ DMV, as it is illegal to drive with expired or no license in the state of New Jersey.

The family is claiming their 9-year-old student was pushed, left out in the cold by school staff members and bullied by other students at Fiske. Mar 19, 2016  1.Print the free eye chart on regular 8 1/2 x 11-inch paper 2.Tack or tape the chart to a windowless wall in a well-lit room at eye level 3.Measure ten feet from the wall.

For more information: Replace a Lost, Stolen, or Mutilated New Jersey Driver’s License or ID Card

How can I obtain a copy of the driver manual?
The New Jersey Driver Manual is a valuable resource when preparing for the DMV drivers exams as all the questions in the test are based on information provided in the manual. The NJ Driver Manual offers information about licenses and vehicle ownership, as well as traffic laws and regulations. You can download the NJ Driver Manual from the State of New Jersey Motor Vehicle Commission website.

For more information: How to Apply for a New Driver’s License in New Jersey

How can I renew my drivers license?
A New Jersey drivers license is good for four years and must be renewed before it expires in person or by mail.

If you are eligible to renew by mail, the NJ MVC will send you “Skip the Trip” package which outlines all the requirements you must meet. If not, you will be required to make an in-person visit at your local NJ DMV and provide documents that satisfy the 6 Point ID verification requirements. You must also pay $24 renewal fee.

Additionally, the NJ DMV requires drivers to undergo periodic vision tests when renewing drivers license.

For more information: How to Renew a Driver’s License in New Jersey

How do I apply for a drivers license?
In order to be eligible to apply for a drivers license in New Jersey, you must have held a probationary drivers license for at least one year. To get your unrestricted NJ drivers license, visit your local MVC office and complete the following steps:
  1. Present your provisional license
  2. Submit documents that meet the 6 Point ID Verification requirements
  3. Provide payment for the $24 new driver’s license fee
  4. Have your photo taken

If you are moving to NJ, you can transfer your out-of-state drivers license within 60 days from moving. Out-of-state learners permits cannot be transferred to New Jersey, teen drivers must complete the stages of the New Jersey GDL program in order to obtain a NJ unrestricted driving license.

For more information: How to Apply for a New Driver’s License in New Jersey

How do I get a hardship license?
The state of New Jersey does not issue any temporary, provisional or work licenses which permit the driver to operate a motor vehicle during certain hours of the day, or for the purpose of traveling to work.

NJ state laws do not allow the issuance of a hardship license after a DWI conviction or any violation that has resulted in a suspension on your drivers license. Moreover, driving on a suspended license incurs harsh penalties in New Jersey, such as five years of jail time.

For more information: Suspended License Information for New Jersey

How do I get a replacement license if I am in the military and stationed elsewhere?
If you are a military member on active duty outside of New Jersey or the country, you are allowed to apply for a duplicate drivers license by mail. To do so, send the following to the NJ MVC:
  • A letter with information why you can’t come in-person to get a duplicate drivers license, including how long you expect to be gone
  • A completed Application for Driver’s License (Form BA-208)
  • Check or money order for $11 made out to to the NJ MVC
  • Documents that meet the 6 points identification verification requirements
  • Current out-of-state or out-of-country address

Mail the package to the following address:

Motor Vehicle Commission Government/Out-of-State Unit P.O. Box 016 Trenton, New Jersey 08666-0016

For more information: Replace a Lost, Stolen, or Mutilated New Jersey Driver’s License or ID Card

How do I know if my license was mailed after it has been restored?
The best way to find out if your drivers license has been mailed out is by contacting the New Jersey Motor Vehicle Commission, directly. To do so, call their toll-free number at 888-486-3339.

If you need same day reinstatement of your driving license, make an in-person visit to any MVC office.

For more information: Suspended License Information for New Jersey

How do I obtain a duplicate license?
Drivers in New Jersey can apply for a drivers license replacement if their license has been lost, stolen, destroyed or damaged beyond legibility. To replace a New Jersey driving license, visit your local NJ DMV and follow these steps:
  1. Provide documents that satisfy the 6 Point ID Verification requirements
  2. Complete an Application for Driver’s License (Form BA-208)
  3. Pay the $11 fee

You may apply for a duplicate drivers license by mail, but only if you meet the requirements.

For more information: Replace a Lost, Stolen, or Mutilated New Jersey Driver’s License or ID Card

Biography of imam qurtubi pdf. PDFROMAN URDU Musalmaan Ka Aqeedah kitab o sunnat ki raushni mein by shaykh Muhammad bin Jamil Zeno 1.05 MB 1 file(s) PDF Seerah of Prophet Muhammad by Imam Anwar al Awlaki (2nd Ed) Complete Transcript. Biography of Imam al - Qurthubi, ince childhood, he lived in a simple family environment. His father was a farmer who is often busy on his farm. He grew up with his father for about 15 years, rather than the year 580 H to 595 H. PDF The Biography of Muawiyah Ibn Sufyan (May Allah be pleased with him) The book is all about the story of first Caliph of Banu Umayyah. Muawiyah hailed from a noble trading family of Mecca (Makkah) and became the first caliph after the four rightly guided caliphs. A short biography of the author: Imam Qurtubi is Muhammad ibn Ahmad ibn Abu Bakr ibn Farah, Abu Abdullah al-Ansari al-Qurtubi, of Cordova (in present day Spain). A Maliki scholar and hadith specialist, he was one of the greatest Imams of Koranic exegesis.

How do I reinstate after a revocation as a Habitual Traffic Offender?
A habitual offender is defined as a New Jersey driver whose drivers license has been suspended three times in three years. Reinstating a suspended drivers license as a Habitual Traffic Offender is the same as restoring a suspended license after your first offence.

Drivers must meet all the restoration requirements listed on the suspension notice, pay the restoration fee and wait to receive a restoration notice in the mail in order to obtain a reinstated driving license.

Keep in mind that you might be faced with stricter penalties and higher fees when reinstating a suspended drivers license as a habitual offender.

For more information: Suspended License Information for New Jersey

How do I reinstate my suspended license?
Drivers in NJ are eligible to reinstate a suspended drivers license once all the restoration requirements, as listed on a NJ suspension notice, have been met. After your suspension period expires and all the conditions have been satisfied, the NJ MVC will mail you a Notice of Restoration and you will be required to pay the $100 restoration fee.

Once you pay the applicable fee, visit a local NJ DMV office and present documents that satisfy the 6 Point ID requirements. A restored NJ driving license will be issued to you that same day.

For more information: Suspended License Information for New Jersey

How do I renew my license if I am in the military and stationed elsewhere?
If you are a military member who is on active military duty or a New Jersey National Guard and Reserve member, your NJ drivers license will be automatically extended until you are fully discharged.

If you are demobilized, you will be required to renew your expired drivers license within two weeks of the date you were demobilized. Make sure you carry the extension letter and documents proving you are on active duty when driving your vehicle.

For more information: How to Renew a Driver’s License in New Jersey

How do I replace my license if I am a non-U.S. citizen?
The process to replace driving license in New Jersey is the same for US and non-US citizens. The major difference lies in the documents you need to provide to meet the 6-point ID program requirements. If you are not a US citizen, you must provide the following documents:
  • One primary proof of identity:
    • Foreign passport with INS or USCIS verification
    • Foreign passport with valid Form I-551 stamp
    • Current alien registration card
    • Refugee travel document (Form I-571)
  • One secondary proof of identity:
    • Current photo employment authorization card
  • Valid Social Security card
  • Proof of NJ residency:
    • Utility bills
    • Bank statements

Keep in mind that the documents presented must be original forms, as the NJ MVC does not accept copies. You will also be required to complete the drivers license replacement application form and pay the applicable fee. You can replace your drivers license in person at a New Jersey DMV location or by mail if you are currently residing out of state.

For more information: Replace a Lost, Stolen, or Mutilated New Jersey Driver’s License or ID Card

How do I replace my license if I lost it out of state?
Drivers in New Jersey must meet the following requirements in order to be eligible to request a drivers license replacement by mail:
  • Applicants drivers license cannot have been lost for over three years.
  • Applicant cannot be currently living in the state of NJ (the DMV does not send temporary permits to NJ addresses).
  • Applicant must be a citizen of the US.
  • Applicant cannot be a CDL holder.
  • Applicant must have a valid New Jersey drivers license.

Also note that only non-digital licenses can be replaced. If your license has a digital picture, you cannot apply for replacement, but instead you must renew your NJ drivers license.

If you meet the aforementioned requirements, send the following documents to the NJ MVC to replace lost drivers license:

  • A letter with information why you can’t come in-person to get a duplicate drivers license, including how long you expect to be gone.
  • A completed Application for Driver’s License (Form BA-208).
  • A check or money order for $11 made out to to the NJ MVC.
  • Documents that meet the 6 points identification verification requirements
  • Current out-of-state or out-of-country address.

For more information: Replace a Lost, Stolen, or Mutilated New Jersey Driver’s License or ID Card

How does the DMV check my vision?
The vision test is administered at New Jersey MVC offices across the state. During the test, a DMV representative will ask you to read a line on an eye chart with both of your eyes open and then another line with each eye individually.

The NJ MVC requires drivers to have 20/50 visual acuity in at least one eye with or without corrective lenses. If a driver fails the vision exam, he/she must visit their doctor and complete a ST-14 form.

For more information: How to Apply for a New Driver’s License in New Jersey

How long will I have a suspended license for not taking a chemical test?
New Jersey has implemented the “implied consent” law, which means that you must take a breathalyzer test if pulled over by the NJ police. Drivers who refuse to undergo a chemical test, will be detained and brought to a hospital where they will have to submit a blood sample for analysis.

The length of your suspension for refusing to take or failing a blood test ranges from seven to 12 months depending on the number of times you have been convicted of a DUI/DWI.

For more information: Suspended License Information for New Jersey

How soon can I renew my driver’s license?
In New Jersey, drivers are allowed to renew an expired NJ driving license as early as six months before it expires. The NJ Motor Vehicle Commission will send you a notice in the mail at least ninety days before your license expires, reminding you to renew your drivers license.

If you are eligible to renew by mail, the NJ MVC will send you a “Skip the Trip” package, which outlines all the requirements you must meet. If not, drivers must make an in-person visit at a local NJ DMV and provide documents that satisfy the 6 Point ID verification requirements.

For more information: How to Renew a Driver’s License in New Jersey

If I file a police report, will my replacement fee be waived?
If you have provided a police report when requesting a drivers license replacement, is to the discretion of the New Jersey MVC if your duplicate drivers license fee will be waived or not.

In any case, NJ state laws require you to file a police report if your drivers license is lost or stolen, as this will protect you from possible fraudulent activity and identity theft.

For more information: Replace a Lost, Stolen, or Mutilated New Jersey Driver’s License or ID Card

If my drivers license has been lost or stolen, how can I get it flagged with the DMV?
The DMV might flag your license number if another driver is pulled over by the police and presents your driving license as their own. Also, a DMV clerk might suggest that you should apply for a duplicate license. However, to be on the safe side, immediately report your drivers license as lost or stolen at your local NJ DMV office.

For more information: Replace a Lost, Stolen, or Mutilated New Jersey Driver’s License or ID Card

Is there an age limit to renew your license?
No, there is no age limit to renew a NJ drivers license. The same rules and regulations in the drivers license renewal process apply for NJ drivers of all ages. The NJ DMV allows drivers to renew expired drivers license by mail or in person at a New Jersey MVC locations across the state.

For more information: How to Renew a Driver’s License in New Jersey

What address will my replacement license be mailed to?
The New Jersey Motor Vehicle Commission does not mail duplicate drivers licenses to mailing addresses. Residents must obtain a driving license replacement in person at a local NJ MVC office.

NJ residents who are temporarily out-of-state and meet the requirements to replace a lost drivers license by mail, will receive their temporary license at the out-of-state or out-of-country address they provide in the application form.

For more information: Replace a Lost, Stolen, or Mutilated New Jersey Driver’s License or ID Card

What are requirements for seniors to renew a driving license?
The same regulations that apply to all NJ drivers when renewing their licenses, apply to senior drivers as well. A New Jersey drivers license is good for four years and must be renewed before it expires in person or by mail. If you have a valid NJ license to drive, you can renew it up to six months before it expires.

The NJ MVC will send you a renewal notice, which outlines all the requirements you must meet to renew drivers license as well as specify if you are eligible to renew by mail. If not, you must visit a local NJ DMV and provide documents that meet the 6 Point ID verification requirements. You must also pay the $24 renewal fee.

For more information: How to Apply for a New Driver’s License in New Jersey

What are some violations that may constitute in a suspended license?
There are several reasons why the New Jersey MVC would suspend your driving privileges. Some of the reasons that could result in a drivers license suspension are listed below:
  • Accumulating over 12 points on your NJ driving record
  • Being involved in a fatal accident or vehicular homicide
  • Reckless driving
  • Failure to appear in court
  • Failure to present proof of car insurance
  • Driving on a suspended drivers license

For more information: Suspended License Information for New Jersey

What are the consequences of driving with a suspended license?
The penalties of driving with a suspended drivers license imposed by the New Jersey Motor Vehicle Commission, depend on the severity of the offence and the number of times the driver has been convicted of it. The fines and penalties implemented in New Jersey vary from points being added to your NJ drivers record to a license suspension. However, driving on a suspended drivers license will lead to harsher penalties, such as 5 years imprisonment in a correctional facility.

For more information: Suspended License Information for New Jersey

What are the fees for a duplicate drivers license?
The fee to obtain a duplicate driving license in New Jersey is $11. Applicants must provide payment for the replacement fee upon visiting NJ MVC offices. Check your local NJ DMV location for acceptable methods of payment.

If you are eligible to replace lost drivers license by mail, provide a check or money order for the $11 replacement fee and send it to the NJ DMV along with the completed application form and necessary documents.

For more information: Replace a Lost, Stolen, or Mutilated New Jersey Driver’s License or ID Card

What are the reinstatement fees I will have to pay for a suspended license?
The basic restoration fee drivers in NJ need to pay to reinstate a suspended drivers license is $100. However, other reinstatement fees may apply, such as an annual surcharge or court fees, depending on the reason for your suspension.

There are several ways to pay the NJ restoration fees, which include:

  • Online
  • In person
  • By mail

For more information: Suspended License Information for New Jersey

What are the suspensions eligible for a hardship license?
The state of New Jersey does not have any temporary, provisional or work licenses, which allow the driver to operate a motor vehicle during certain hours of the day, or for the purpose of traveling to work.

NJ state laws do not allow the issuance of a hardship license after a DWI conviction or any violation that has resulted in a suspension on your drivers license. Moreover, driving on a suspended license incurs harsh penalties in New Jersey, such as five years of jail time.

For more information: Suspended License Information for New Jersey

What do I need to do to get my driver’s license if I fail the driving test?
If you fail the driving exam required in order to obtain a NJ drivers license, you must wait for a minimum of two weeks before being allowed to re-take the test. If you fail the driving test more than twice, you will be required to wait at least six months before re-taking the NJ skills test.

When taking the DMV drivers test, make sure you are accompanied by a licensed driver and present forms of ID as required under the 6 Point ID Verification Program. Applicants must also provide a fully registered vehicle with current inspection sticker and insurance card to use when taking the drivers exam.

For more information: How to Apply for a New Driver’s License in New Jersey

What documents do I need to get my drivers license?
Drivers license applicants must meet the New Jersey Motor Vehicle Commission 6 Point ID Verification requirements in order to obtain a license to drive. To do so, new drivers must present the following documents:
  • One primary proof of identity
  • One secondary proof of identity
  • Proof of valid Social Security Number
  • Proof of address

For more help regarding the selection of documents and satisfying the requirements, visit the MVC website. If you have an out-of-state drivers license, you must surrender it to the NJ MVC when applying for a new driving license.

For more information: How to Apply for a New Driver’s License in New Jersey

What documents do I need to present to renew my drivers license?
There are two ways to renew a New Jersey drivers license: in person or by mail. If you are eligible to renew by mail, the NJ MVC will send you a “Skip the Trip” package, which outlines all the requirements you must meet. Applicants will be required to complete the application included in the package and mail it along with a check or money order for the renewal fee to the NJ MVC.

If you are not allowed to renew your expired drivers license by mail, visit a NJ DMV location and provide documents that meet the NJ 6 point ID verification requirements. These include:

  • One primary proof of identity:
    • Current NJ drivers license
    • Birth certificate
    • US passport
  • One secondary proof of identity:
    • Military ID
    • Marriage certificate
  • Proof of valid Social Security Number
  • Proof of address:
    • Utility bills
    • Bank statements

For more information on selecting the right documents and satisfying the requirements of the 6 point ID verification program, visit the MVC website. Also make sure you provide payment of $24 for the renewal fee.

For more information: How to Renew a Driver’s License in New Jersey

What documents will I need to present to replace my drivers license?
To replace a NJ driving license, visit a NJ DMV location and present documents that meet the 6 Point ID Verification requirements, which include:
  • One primary proof of identity:
    • Current NJ drivers license
    • Birth certificate
    • US passport
  • One secondary proof of identity:
    • Military ID
    • Marriage certificate
  • Proof of valid Social Security Number
  • Proof of address:
    • Utility bills
    • Bank statements

For more information on selecting the right documents and satisfying the requirements of the 6 point ID verification program, visit the MVC website. You are also required to complete the Application for Driver’s License (Form BA-208). This form is not available for download and can only be obtained at a New Jersey DMV office.

For more information: Replace a Lost, Stolen, or Mutilated New Jersey Driver’s License or ID Card

What languages is the written test available in?
The New Jersey written drivers test is available in the following languages:
  • English
  • Arabic
  • Chinese
  • French
  • Spanish
  • Korean
  • Polish
  • Portuguese
  • Russian
  • Japanese

In addition, the New Jersey MVC offers oral tests in English and Spanish. If your native language is not the list, you are allowed to use the services of a state-approved interpreter when taking the DMV drivers exam.

For more information: How to Apply for a New Driver’s License in New Jersey

What penalties will I face if I do not renew my expired drivers license?
Driving with an expired driving license is illegal in New Jersey. Possible penalties include a fine in the amount of $150 and even a drivers license suspension.

Also, keep in mind that if you have not renewed your drivers license within three years after the expiration date, you will have to apply for a new drivers license.

For more information: How to Renew a Driver’s License in New Jersey

What should I do if I have not received my renewed license?
ExamIt usually takes the New Jersey MVC ten business days to process the documentation and payment and issue your renewed NJ drivers license. If your renewed driving license has not arrived by that time, contact the NJ DMV for more details on the delivery status of your license to drive.

For more information: How to Renew a Driver’s License in New Jersey

What tests will I need to take for my drivers license?
The New Jersey Motor Vehicle Commission does not require you to take any tests when applying for drivers license. However, you must have held a learners permit for at least six months and a probationary drivers license for at least one year before being eligible for a new NJ driving license. First-time drivers applying for a learners permit must take the following tests:
  • Vision test
  • Written knowledge test
  • Driving exam

For more information: How to Apply for a New Driver’s License in New Jersey

What type of noncommercial license will I need?
The NJ Motor Vehicle Commission issues the following types of non-commercial drivers licenses:
  • Learners permits and restricted licenses for first-time drivers under the Graduated Drivers License program, which include:
    • Student learners permit
    • Examination permit
    • Probationary license
  • Basic automobile license (class D), which is issued to non-commercial drivers who are 18 years of age and older.
  • Motorcycle license (class E) , which can be issued as a separate license or endorsement on your existing NJ driving license.

For more information: How to Apply for a New Driver’s License in New Jersey

When can I reinstate my license after it has been suspended?
Drivers can reinstate a suspended drivers license if they have met all the restoration requirements as listed on a NJ suspension notice. After a suspension period expires and all the conditions have been satisfied, the NJ MVC will send you a Notice of Restoration. Drivers will then be required to pay the $100 restoration fee.

For more information: Suspended License Information for New Jersey

When will I need to renew my license?
The New Jersey license to drive is valid for four years and must be renewed before its expiration date in person or by mail. The NJ Motor Vehicle Commission allows you to renew up to six months before your driving license expires. In addition, you will receive a renewal notice from the New Jersey MVC at least ninety days before your license expires reminding you to renew your drivers license.

Drivers can renew their NJ drivers license by mail or in person at a local NJ MVC.

For more information: How to Renew a Driver’s License in New Jersey

When will I receive a replacement license after applying?
Drivers applying for a drivers license replacement in person will be issued a duplicate driving license immediately. Drivers applying by mail must wait a minimum of ten business days for the New Jersey Motor Vehicle Commission to process the payment and the documentation provided.

If your drivers license copy has not arrived at your out-of-state or out-of-country address within that time, contact the New Jersey DMV to check on the delivery status of your driver s license replacement.

For more information: Replace a Lost, Stolen, or Mutilated New Jersey Driver’s License or ID Card

Where can I check to see if my license is suspended?
NJ drivers can check their New Jersey driving record for information on their drivers license status. Drivers can also check if they have met all the reinstatement requirements by visiting a local NJ MVC office or regional service center. To find the nearest NJ MVC, you can use our DMV locator.

For more information: Suspended License Information for New Jersey

Where can I get a new drivers license?
To obtain a new drivers license in New Jersey, drivers must visit a MVC Driver Testing Center. Upon arrival, drivers need to present certain documents required under the 6 Point ID Verification system as well as a current NJ provisional drivers license. Additionally, applicants must pay the $24 new drivers license fee. Check with the DMV near you for accepted methods of payment.

For more information: How to Apply for a New Driver’s License in New Jersey

Will I be required to take a vision test for a drivers license renewal?
Not all drivers have to pass a vision test when renewing expired driving license in New Jersey. However, the NJ MVC administers periodic vision exams for driving license renewals.

The NJ MVC requires drivers to have 20/50 visual acuity in at least one eye with or without corrective lenses. If a driver fails the vision exam, he/she must visit their doctor and complete a ST-14 form.

For more information: How to Renew a Driver’s License in New Jersey

Will I get a drivers license renewal notice in the mail?
Yes, the NJ Motor Vehicle Commission will send you a notice in the mail at least ninety days before your license expires reminding you to renew your drivers license.

If you are eligible to renew by mail, the NJ MVC will also send you a “Skip the Trip” package, which outlines all the requirements you must meet. If not, you need to make an in-person visit at your local NJ DMV and provide documents that satisfy the 6 Point ID verification requirements.

However, keep in mind that even if you do not receive a renewal notice in the mail, it is your responsibility to check the status of your NJ driving license. To do so, you can request a copy of your New Jersey drivers record.

For more information: How to Renew a Driver’s License in New Jersey

Color blindness
Other namesColour blindness, color deficiency, impaired color vision[1]
Example of an Ishihara color test plate. With properly configured computer displays, people with normal vision should see the number '74'. Many people who are color blind see it as '21', and those with total color blindness may not see any numbers.
SpecialtyOphthalmology
SymptomsDecreased ability to see colors[2]
DurationLong term[2]
CausesGenetic (inherited usually X-linked)[2]
Diagnostic methodIshihara color test[2]
TreatmentAdjustments to teaching methods, mobile apps[1][2]
FrequencyRed–green: 8% males, 0.5% females (Northern European descent)[2]

Color blindness, also known as color vision deficiency, is the decreased ability to see color or differences in color.[2] Simple tasks such as selecting ripe fruit, choosing clothing, and reading traffic lights can be more challenging.[2] Color blindness may also make some educational activities more difficult.[2] However, problems are generally minor, and most people find that they can adapt.[2] People with total color blindness (achromatopsia) may also have decreased visual acuity and be uncomfortable in bright environments.[2]

The most common cause of color blindness is an inherited problem in the development of one or more of the three sets of color-sensing cones in the eye.[2] Males are more likely to be color blind than females, as the genes responsible for the most common forms of color blindness are on the X chromosome.[2] As females have two X chromosomes, a defect in one is typically compensated for by the other, therefore females can be carriers.[2] Males only have one X chromosome and therefore express the genetic disorder.[2] Color blindness can also result from physical or chemical damage to the eye, optic nerve or parts of the brain.[2] Diagnosis is typically with the Ishihara color test; however, a number of other testing methods, including genetic testing, also exist.[2][3]

There is no cure for color blindness.[2] Diagnosis may allow a person's teacher to change their method of teaching to accommodate the decreased ability to recognize colors.[1] Special lenses may help people with red–green color blindness when under bright conditions.[2] There are also mobile apps that can help people identify colors.[2]

Red–green color blindness is the most common form, followed by blue–yellow color blindness and total color blindness.[2] Red–green color blindness affects up to 8% of males and 0.5% of females of Northern European descent.[2] The ability to see color also decreases in old age.[2] Being color blind may make people ineligible for certain jobs in certain countries.[1] This may include being a pilot, train driver, crane operator, and working in the armed forces.[1][4] The effect of color blindness on artistic ability, however, is controversial.[1][5] The ability to draw appears to be unchanged, and a number of famous artists are believed to have been color blind.[1][6]

  • 2Causes
  • 4Diagnosis
    • 4.1Classification
  • 5Management
  • 8Society and culture
    • 8.6Rights of the color blind

Signs and symptoms[edit]

Simulation of the normal (above) and dichromatic (below) perception of red and green apples
Horizontal traffic light in Halifax, Nova Scotia, Canada

In almost all cases, color blind people retain blue–yellow discrimination, and most color-blind individuals are anomalous trichromats rather than complete dichromats. In practice, this means that they often retain a limited discrimination along the red–green axis of color space, although their ability to separate colors in this dimension is reduced. Color blindness very rarely refers to complete monochromatism.[7]

Dichromats often confuse red and green items. For example, they may find it difficult to distinguish a Braeburn apple from a Granny Smith or red from green of traffic lights without other clues—for example, shape or position. Dichromats tend to learn to use texture and shape clues and so may be able to penetrate camouflage that has been designed to deceive individuals with normal color vision.[8]

Colors of traffic lights are confusing to some dichromats as there is insufficient apparent difference between the red/amber traffic lights and sodium street lamps; also, the green can be confused with a grubby white lamp. This is a risk on high-speed undulating roads where angular cues cannot be used. British Rail color lamp signals use more easily identifiable colors: The red is blood red, the amber is yellow and the green is a bluish color. Most British road traffic lights are mounted vertically on a black rectangle with a white border (forming a 'sighting board') and so dichromats can more easily look for the position of the light within the rectangle—top, middle or bottom. In the eastern provinces of Canada horizontally mounted traffic lights are generally differentiated by shape to facilitate identification for those with color blindness.[citation needed] In the United States, this is not done by shape but by position, as the red light is always on the left if the light is horizontal, or on top if the light is vertical. However, a lone flashing light (e.g. red for stop, yellow for caution) is still problematic.

  • Normal sight

  • Deuteranopia sight

  • Tritanopia sight

  • Monochromacy sight

Causes[edit]

Color vision deficiencies can be classified as acquired or inherited.

  • Acquired: Diseases, drugs (e.g., Plaquenil), and chemicals such as styrene[9] or organic solvents[10] may cause color blindness.[11][12]
  • Inherited: There are three types of inherited or congenital color vision deficiencies: monochromacy, dichromacy, and anomalous trichromacy.
    • Monochromacy, also known as 'total color blindness', is the lack of ability to distinguish colors (and thus the person views everything as if it were on a black and white television); caused by cone defect or absence. Monochromacy occurs when two or all three of the cone pigments are missing and color and lightness vision is reduced to one dimension.
      • Rod monochromacy (achromatopsia) is an exceedingly rare, nonprogressive inability to distinguish any colors as a result of absent or nonfunctioning retinal cones. It is associated with light sensitivity (photophobia), involuntary eye oscillations (nystagmus), and poor vision.
      • Cone monochromacy is a rare total color blindness that is accompanied by relatively normal vision, electroretinogram, and electrooculogram. Cone monochromacy can also be a result of having more than one type of dichromatic color blindness. People who have, for instance, both protanopia and tritanopia are considered to have cone monochromacy. Since cone monochromacy is the lack of/damage of more than one cone in retinal environment, having two types of dichromacy would be an equivalent.
    • Dichromacy is hereditary. Protanopia and deuteranopia are hereditary and sex-linked, affecting predominantly males.
      • Protanopia is caused by the complete absence of red retinal photoreceptors. Protans have difficulties distinguishing between blue and green colors and also between red and green colors. It is a form of dichromatism in which the subject can only perceive light wavelengths from 400 nm to 650 nm, instead of the usual 700 nm. Pure reds cannot be seen, instead appearing black; purple colors cannot be distinguished from blues; more orange-tinted reds may appear as dim yellows, and all orange–yellow–green shades of too long a wavelength to stimulate the blue receptors appear as a similar yellow hue. It is present in 1% of males.
      • Deuteranopia affects hue discrimination in a similar way to protanopia, but without the dimming effect. Again, it is found in about 1% of the male population.[13]
      • Tritanopia is a very rare color vision disturbance in which only the red and the green cone pigments are present, with a total absence of blue retinal receptors. Blues appear greenish, yellows and oranges appear pinkish, and purple colors appear deep red. It is related to chromosome 7; thus unlike protanopia and deuteranopia, tritanopia and tritanomaly are not sex-linked traits and can be acquired rather than inherited and can be reversed in some cases.
    • Anomalous trichromacy is a common type of inherited color vision deficiency, occurring when one of the three cone pigments is altered in its spectral sensitivity.
      • Protanomaly is a mild color vision defect in which an altered spectral sensitivity of red retinal receptors (closer to green receptor response) results in poor red–green hue discrimination. It is hereditary, sex-linked, and present in 1% of males. In contrast to other defects, in this case the L-cone is present but malfunctioning, whereas in protanopia the L-cone is completely missing.[14]
      • Deuteranomaly, caused by a similar shift in the green retinal receptors, is by far the most common type of color vision deficiency, mildly affecting red–green hue discrimination in 5% of European males. It is hereditary and sex-linked. In contrast to deuteranopia, the green-sensitive cones are not missing but malfunctioning.[15]
      • Tritanomaly is a rare, hereditary color vision deficiency affecting blue–green and yellow–red/pink hue discrimination. It is related to chromosome 7.[16] In contrast to tritanopia, the S-cone is malfunctioning but not missing.[17]

Genetics[edit]

X-linked recessive inheritance

Color blindness is typically an inherited genetic disorder. It is most commonly inherited from mutations on the X chromosome, but the mapping of the human genome has shown there are many causative mutations—mutations capable of causing color blindness originate from at least 19 different chromosomes and 56 different genes (as shown online at the Online Mendelian Inheritance in Man (OMIM)).

Two of the most common inherited forms of color blindness are protanomaly (and, more rarely, protanopia—the two together often known as 'protans') and deuteranomaly (or, more rarely, deuteranopia—the two together often referred to as 'deutans').[18]Both 'protans' and 'deutans' (of which the deutans are by far the most common) are known as 'red–green color-blind'. They comprise about 8% of human males and 0.6% of females of Northern European ancestry.[19]

Some of the inherited diseases known to cause color blindness are:

  • achromatopsia (also called rod monochromatism, stationary cone dystrophy or cone dysfunction syndrome)
  • blue cone monochromatism (also called blue cone monochromacy or X-linked achromatopsia)
  • retinitis pigmentosa (initially affects rods but can later progress to cones and therefore color blindness).

Inherited color blindness can be congenital (from birth), or it can commence in childhood or adulthood. Depending on the mutation, it can be stationary, that is, remain the same throughout a person's lifetime, or progressive. As progressive phenotypes involve deterioration of the retina and other parts of the eye, certain forms of color blindness can progress to legal blindness, i.e. an acuity of 6/60 (20/200) or worse, and often leave a person with complete blindness.

Color blindness always pertains to the cone photoreceptors in retinas, as it is the cones that detect the color frequencies of light.

About 8% of males, and 0.6% of females, are red–green color blind in some way or another, whether it is one color, a color combination, or another mutation.[20] Males are at a greater risk of inheriting an X-linked mutation because males only have one X chromosome (XY, with the Y chromosome carrying altogether different genes from the X chromosome), and females have two (XX); if a woman inherits a normal X chromosome in addition to the one that carries the mutation, she will not display the mutation. Men do not have a second X chromosome to override the chromosome that carries the mutation. If 8% of variants of a given gene are defective, the probability of a single copy being defective is 8%, but the probability that two copies are both defective is 0.082, i.e. 0.64%.

Other causes[edit]

Other causes of color blindness include brain or retinal damage caused by accidents and other traumas which produce swelling of the brain in the occipital lobe, and damage to the retina caused by exposure to ultraviolet light (wavelengths 10 to 300 nm). Damage often presents itself later in life.

Color blindness may also present itself in the range of degenerative diseases of the eye, such as age-related macular degeneration, and as part of the retinal damage caused by diabetes. Vitamin A deficiency may also cause color blindness.[21]

Some subtle forms of color blindness may be associated with chronic solvent-induced encephalopathy (CSE), caused by long-time exposure to solvent vapors.[22]

Red–green color blindness can be caused by ethambutol,[23] a drug used in the treatment of tuberculosis.

Mechanism[edit]

The typical human retina contains two kinds of light cells: the rod cells (active in low light) and the cone cells (active in normal daylight). Normally, there are three kinds of cone cells, each containing a different pigment, which are activated when the pigments absorb light. The spectral sensitivities of the cones differ; one is most sensitive to short wavelengths, one to medium wavelengths, and the third to medium-to-long wavelengths within the visible spectrum, with their peak sensitivities in the blue, green, and yellow–green regions of the spectrum, respectively. The absorption spectra of the three systems overlap, and combine to cover the visible spectrum. These receptors are known as short (S), medium (M), and long (L) wavelength cones, but are also often referred to as blue, green, and red cones, although this terminology is inaccurate.[24]

The receptors are each responsive to a wide range of wavelengths. For example, the long wavelength 'red' receptor has its peak sensitivity in the yellow–green, some way from the red end (longest wavelength) of the visible spectrum. The sensitivity of normal color vision actually depends on the overlap between the absorption ranges of the three systems: different colors are recognized when the different types of cone are stimulated to different degrees. Red light, for example, stimulates the long wavelength cones much more than either of the others, and reducing the wavelength causes the other two cone systems to be increasingly stimulated, causing a gradual change in hue.

Many of the genes involved in color vision are on the X chromosome, making color blindness much more common in males than in females because males only have one X chromosome, while females have two. An estimated 2–3% of women have two slightly different red colour cones[25] and can be considered tetrachromats. One such woman has been reported to be a true or functional tetrachromat, as she can discriminate colors most other people can't.[26][27]

Diagnosis[edit]

Florida Dmv Eye Test Chart

An Ishihara test image as seen by subjects with normal color vision and by those with a variety of color deficiencies

The Ishihara color test, which consists of a series of pictures of colored spots, is the test most often used to diagnose red–green color deficiencies.[28] A figure (usually one or more Arabic digits) is embedded in the picture as a number of spots in a slightly different color, and can be seen with normal color vision, but not with a particular color defect. The full set of tests has a variety of figure/background color combinations, and enable diagnosis of which particular visual defect is present. The anomaloscope, described above, is also used in diagnosing anomalous trichromacy.

Position yourself about 75cm from your monitor so that the colour test image you are looking at is at eye level, read the description of the image and see what you can see!! It is not necessary in all cases to use the entire set of images. In a large scale examination the test can be simplified to six tests; test, one of tests 2 or 3, one of tests 4, 5, 6, or 7, one of tests 8 or 9, one of tests 10, 11, 12, or 13 and one of tests 14 or 15.[This quote needs a citation]

Ohio Drivers Eye Exam Chart For Driver S License

Because the Ishihara color test contains only numerals, it may not be useful in diagnosing young children, who have not yet learned to use numbers. In the interest of identifying these problems early on in life, alternative color vision tests were developed using only symbols (square, circle, car).

Besides the Ishihara color test, the US Navy and US Army also allow testing with the Farnsworth Lantern Test. This test allows 30% of color deficient individuals, whose deficiency is not too severe, to pass.

Another test used by clinicians to measure chromatic discrimination is the Farnsworth–Munsell 100 hue test. The patient is asked to arrange a set of colored caps or chips to form a gradual transition of color between two anchor caps.[29]

The HRR color test (developed by Hardy, Rand, and Rittler) is a red–green color test that, unlike the Ishihara, also has plates for the detection of the tritan defects.[30]

Most clinical tests are designed to be fast, simple, and effective at identifying broad categories of color blindness. In academic studies of color blindness, on the other hand, there is more interest in developing flexible tests to collect thorough datasets, identify copunctal points, and measure just noticeable differences.[31]

Classification[edit]

These color charts show how different colorblind people see compared to a person with normal color vision.

Types of color blindness and the terms used

Cone
system
RedGreenBlue
N=normal
A=anomalous
NANANA
1Normal visionTrichromatNormal
2ProtanomalyAnomalous TrichromatPartially color blindRed–green
3ProtanopiaDichromatPartially color blindRed–green
4DeuteranomalyAnomalous TrichromatPartially color blindRed–green
5DeuteranopiaDichromatPartially color blindRed–green
6TritanomalyAnomalous TrichromatPartially color blindBlue–yellow
7TritanopiaDichromatPartially color blindBlue–yellow
8AchromatopsiaMonochromatTotally color blind
9Tetrachromat
10

Based on clinical appearance, color blindness may be described as total or partial. Total color blindness is much less common than partial color blindness.[32] There are two major types of color blindness: difficulty distinguishing between red and green, and difficulty distinguishing between blue and yellow.[33][34]

Immunofluorescent imaging is a way to determine red–green color coding. Conventional color coding is difficult for individuals with red–green color blindness (protanopia or deuteranopia) to discriminate. Replacing red with magenta or green with turquoise improves visibility for such individuals.[35]

The different kinds of inherited color blindness result from partial or complete loss of function of one or more of the three different cone systems. When one cone system is compromised, dichromacy results. The most frequent forms of human color blindness result from problems with either the middle (green) or long (red) wavelength sensitive cone systems, and make it hard to discriminate reds, yellows, and greens from one another. They are collectively referred to as 'red–green color blindness', though the term is an over-simplification and is somewhat misleading. Other forms of color blindness are much more rare. They include problems in discriminating blues from greens and yellows from reds/pinks, and the rarest form of all, complete color blindness or monochromacy, where one cannot distinguish any color from grey, as in a black-and-white movie or photograph.

Protanopes, deuteranopes, and tritanopes are dichromats; that is, they can match any color they see with some mixture of just two primary colors (in contrast to those with normal sight (trichromats) who can distinguish three primary colors). Dichromats usually know they have a color vision problem, and it can affect their daily lives. Out of the male population, 2% have severe difficulties distinguishing between red, orange, yellow, and green. (Orange and yellow are different combinations of red and green light.) Colors in this range, which appear very different to a normal viewer, appear to a dichromat to be the same or a similar color. The terms protanopia, deuteranopia, and tritanopia come from Greek, and respectively mean 'inability to see (anopia) with the first (prot-), second (deuter-), or third (trit-) [cone]'.

Anomalous trichromacy is the least serious type of color deficiency.[36] People with protanomaly, deuteranomaly, or tritanomaly are trichromats, but the color matches they make differ from the normal. They are called anomalous trichromats. In order to match a given spectral yellow light, protanomalous observers need more red light in a red/green mixture than a normal observer, and deuteranomalous observers need more green. From a practical standpoint though, many protanomalous and deuteranomalous people have very little difficulty carrying out tasks that require normal color vision. Some may not even be aware that their color perception is in any way different from normal.

Protanomaly and deuteranomaly can be diagnosed using an instrument called an anomaloscope, which mixes spectral red and green lights in variable proportions, for comparison with a fixed spectral yellow. If this is done in front of a large audience of males, as the proportion of red is increased from a low value, first a small proportion of the audience will declare a match, while most will see the mixed light as greenish; these are the deuteranomalous observers. Next, as more red is added the majority will say that a match has been achieved. Finally, as yet more red is added, the remaining, protanomalous, observers will declare a match at a point where normal observers will see the mixed light as definitely reddish.

Red–green color blindness[edit]

Protanopia, deuteranopia, protanomaly, and deuteranomaly are commonly inherited forms of red–green color blindness which affect a substantial portion of the human population. Those affected have difficulty with discriminating red and green hues due to the absence or mutation of the red or green retinal photoreceptors.[18][37] It is sex-linked: genetic red–green color blindness affects males much more often than females, because the genes for the red and green color receptors are located on the X chromosome, of which males have only one and females have two. Females (XX) are red–green color blind only if both their X chromosomes are defective with a similar deficiency, whereas males (XY) are color blind if their single X chromosome is defective.[38]

The gene for red–green color blindness is transmitted from a color blind male to all his daughters, who are usually heterozygote carriers and are thus unaffected. In turn, a carrier woman has a 50% chance of passing on a mutated X chromosome region to each of her male offspring. The sons of an affected male will not inherit the trait from him, since they receive his Y chromosome and not his (defective) X chromosome. Should an affected male have children with a carrier or colorblind woman, their daughters may be colorblind by inheriting an affected X chromosome from each parent.[38]

Because one X chromosome is inactivated at random in each cell during a woman's development, deuteranomalous heterozygotes (i.e. female carriers of deuteranomaly) may be tetrachromats, because they will have the normal long wave (red) receptors, the normal medium wave (green) receptors, the abnormal medium wave (deuteranomalous) receptors and the normal autosomal short wave (blue) receptors in their retinas.[25][26][27] The same applies to the carriers of protanomaly (who have two types of long wave receptors, normal medium wave receptors, and normal autosomal short wave receptors in their retinas). If, by rare chance, a woman is heterozygous for both protanomaly and deuteranomaly, she could be pentachromatic. This situation could arise if, for instance, she inherited the X chromosome with the abnormal long wave gene (but normal medium wave gene) from her mother who is a carrier of protanomaly, and her other X chromosome from a deuteranomalous father. Such a woman would have a normal and an abnormal long wave receptor, a normal and abnormal medium wave receptor, and a normal autosomal short wave receptor—5 different types of color receptors in all. The degree to which women who are carriers of either protanomaly or deuteranomaly are demonstrably tetrachromatic and require a mixture of four spectral lights to match an arbitrary light is very variable. In many cases it is almost unnoticeable, but in a minority the tetrachromacy is very pronounced.[25][26][27] However, Jameson et al.[39] have shown that with appropriate and sufficiently sensitive equipment it can be demonstrated that any female carrier of red–green color blindness (i.e. heterozygous protanomaly, or heterozygous deuteranomaly) is a tetrachromat to a greater or lesser extent. People in whom deuteranopia or deuteranomaly manifest are sometimes referred to as deutans, those with protanopia or protanomaly as protans.[40]

Since deuteranomaly is by far the most common form of red–green blindness among men of northwestern European descent (with an incidence of 8%) it follows that the proportion of carriers (and of potential deuteranomalous tetrachromats) among the females of that genetic stock is 14.7% (i.e. 92% × 8% × 2), based on the Hardy–Weinberg principle.[38]

Illustration of the distribution of cone cells in the fovea of an individual with normal color vision (left), and a color blind (protanopic) retina. The center of the fovea holds very few blue-sensitive cones.
  • Protanopia (1% of males): Lacking the red cones for long-wavelength sensitive retinal cones, those with this condition are unable to distinguish between colors in the green–yellow–red section of the spectrum. They have a neutral point at a cyan-like wavelength around 492 nm (see spectral color for comparison)—that is, they cannot discriminate light of this wavelength from white. For a protanope, the brightness of red, orange, and yellow are much reduced compared to normal. This dimming can be so pronounced that reds may be confused with black or dark gray, and red traffic lights may appear to be extinguished. They may learn to distinguish reds from yellows primarily on the basis of their apparent brightness or lightness, not on any perceptible hue difference. Violet, lavender, and purple are indistinguishable from various shades of blue because their reddish components are so dimmed as to be invisible. For example, pink flowers, reflecting both red light and blue light, may appear just blue to the protanope. A very few people have been found who have one normal eye and one protanopic eye. These unilateral dichromats report that with only their protanopic eye open, they see wavelengths shorter than neutral point as blue and those longer than it as yellow. This is a rare form of color blindness.
  • Deuteranopia (1% of males): Lacking the green cones for medium-wavelength cones, those affected are again unable to distinguish between colors in the green–yellow–red section of the spectrum. Their neutral point is at a slightly longer wavelength, 498 nm, a more greenish hue of cyan. A deuteranope suffers the same hue discrimination problems as protanopes, but without the abnormal dimming. Purple colors are not perceived as something opposite to spectral colors; all these appear similarly. This form of colorblindness is also known as Daltonism after John Dalton (his diagnosis was confirmed as deuteranopia in 1995, some 150 years after his death, by DNA analysis of his preserved eyeball). Equivalent terms for Daltonism in Romance languages such as daltonismo (Spanish, Portuguese and Italian), daltonisme (French), daltonism (Romanian) and in Slavic languages such as daltonizam (Croatian), daltonizam/далтонизам (Serbian), далтонизам (Macedonian), дальтонизм (Russian), дальтонізм (Ukrainian) and дальтанізм (Belarusian) are still used to describe color blindess in a broad sense or deuteranopia in a more restricted sense. Deuteranopic unilateral dichromats report that with only their deuteranopic eye open, they see wavelengths shorter than neutral point as blue and longer than it as yellow.[41]
  • Protanomaly (1% of males, 0.01% of females):[42] Having a mutated form of the long-wavelength (red) pigment, whose peak sensitivity is at a shorter wavelength than in the normal retina, protanomalous individuals are less sensitive to red light than normal. This means that they are less able to discriminate colors, and they do not see mixed lights as having the same colors as normal observers. They also suffer from a darkening of the red end of the spectrum. This causes reds to reduce in intensity to the point where they can be mistaken for black. Protanomaly is a fairly rare form of color blindness, making up about 1% of the male population. Both protanomaly and deuteranomaly are carried on the X chromosome.
  • Deuteranomaly (most common—6% of males, 0.4% of females):[42] These individuals have a mutated form of the medium-wavelength (green) pigment. The medium-wavelength pigment is shifted towards the red end of the spectrum resulting in a reduction in sensitivity to the green area of the spectrum. Unlike in protanomaly, the intensity of colors is unchanged. The deuteranomalous person is considered 'green weak'. For example, in the evening, dark green cars appear to be black to deuteranomalous people. As with protanomates, deuteranomates are poor at discriminating small differences in hues in the red, orange, yellow, green region of the spectrum. They make errors in the naming of hues in this region because the hues appear somewhat shifted towards green. However, unlike protanomates, deuteranomalous people do not have the loss of 'brightness' problem.

Blue–yellow color blindness[edit]

Those with tritanopia and tritanomaly have difficulty discerning between bluish and greenish hues, as well as yellowish and reddish hues.

Color blindness involving the inactivation of the short-wavelength sensitive cone system (whose absorption spectrum peaks in the bluish-violet) is called tritanopia or, loosely, blue–yellow color blindness. The tritanope's neutral point occurs near a yellowish 570 nm; green is perceived at shorter wavelengths and red at longer wavelengths.[43] Mutation of the short-wavelength sensitive cones is called tritanomaly. Tritanopia is equally distributed among males and females. Jeremy H. Nathans (with the Howard Hughes Medical Institute) demonstrated that the gene coding for the blue receptor lies on chromosome 7, which is shared equally by males and females. Therefore, it is not sex-linked. This gene does not have any neighbor whose DNA sequence is similar. Blue color blindness is caused by a simple mutation in this gene.

  • Tritanopia (less than 1% of males and females): Lacking the short-wavelength cones, those affected see short-wavelength colors (blue, indigo and a spectral violet) greenish and drastically dimmed, some of these colors even as black. Yellow is indistinguishable from pink, and purple colors are perceived as various shades of red. This form of color blindness is not sex-linked.
  • Tritanomaly (equally rare for males and females [0.01% for both]):[42] Having a mutated form of the short-wavelength (blue) pigment. The short-wavelength pigment is shifted towards the green area of the spectrum. This is the rarest form of anomalous trichromacy color blindness. Unlike the other anomalous trichromacy color deficiencies, the mutation for this color blindness is carried on chromosome 7. Therefore, it is equally prevalent in both male and female populations. The OMIM gene code for this mutation is 304000 'Colorblindness, Partial Tritanomaly'.[44]

Total color blindness[edit]

Total color blindness is defined as the inability to see color. Although the term may refer to acquired disorders such as cerebral achromatopsia also known as color agnosia, it typically refers to congenital color vision disorders (i.e. more frequently rod monochromacy and less frequently cone monochromacy).[45][46]

In cerebral achromatopsia, a person cannot perceive colors even though the eyes are capable of distinguishing them. Some sources do not consider these to be true color blindness, because the failure is of perception, not of vision. They are forms of visual agnosia.[46]

Monochromacy is the condition of possessing only a single channel for conveying information about color. Monochromats possess a complete inability to distinguish any colors and perceive only variations in brightness. It occurs in two primary forms:

  1. Rod monochromacy, frequently called achromatopsia, where the retina contains no cone cells, so that in addition to the absence of color discrimination, vision in lights of normal intensity is difficult. While normally rare, achromatopsia is very common on the island of Pingelap, a part of the Pohnpei state, Federated States of Micronesia, where it is called maskun: about 10% of the population there has it, and 30% are unaffected carriers. The island was devastated by a storm in the 18th century (an example of a genetic bottleneck) and one of the few male survivors carried a gene for achromatopsia. The population grew to several thousand before foreign troops introduced diseases to the island in the 1940s.
  2. Cone monochromacy is the condition of having both rods and cones, but only a single kind of cone. A cone monochromat can have good pattern vision at normal daylight levels, but will not be able to distinguish hues. Blue cone monochromacy (X chromosome) is caused by lack of functionality of L and M cones (red and green). It is encoded at the same place as red–green color blindness on the X chromosome. Peak spectral sensitivities are in the blue region of the visible spectrum (near 440 nm). People with this condition generally show nystagmus ('jiggling eyes'), photophobia (light sensitivity), reduced visual acuity, and myopia (nearsightedness).[47] Visual acuity usually falls to the 20/50 to 20/400 range.

Management[edit]

There is generally no treatment to cure color deficiencies. ″The American Optometric Association reports a contact lens on one eye can increase the ability to differentiate between colors, though nothing can make you truly see the deficient color.″[48]

Lenses[edit]

Optometrists can supply colored spectacle lenses or a single red-tint contact lens to wear on the non-dominant eye, but although this may improve discrimination of some colors, it can make other colors more difficult to distinguish. A 1981 review of various studies to evaluate the effect of the X-chrom contact lens concluded that, while the lens may allow the wearer to achieve a better score on certain color vision tests, it did not correct color vision in the natural environment.[49] A case history using the X-Chrom lens for a rod monochromat is reported[50] and an X-Chrom manual is online.[51]

Lenses that filter certain wavelengths of light can allow people with a cone anomaly, but not dichromacy, to see better separation of colors, especially those with classic 'red/green' color blindness. They work by notching out wavelengths that strongly stimulate both red and green cones in a deuter- or protanomalous person, improving the distinction between the two cones' signals. As of 2012, sunglasses that notch out color wavelengths are available commercially.[52]

Apps[edit]

Many mobile applications have been developed to help colorblind people to view colors in a better way. Many applications launch a simulation of colorblindness to allow people with normal vision to understand how people with color blindness see the world.[citation needed]

Epidemiology[edit]

Rates of color blindness[clarification needed][53]
MalesFemales
Dichromacy2.4%0.03%
Protanopia (red deficient: L cone absent)1.3%0.02%
Deuteranopia (green deficient: M cone absent)1.2%0.01%
Tritanopia (blue deficient: S cone absent)0.001%0.03%
Anomalous trichromacy6.3%0.37%
Protanomaly (red deficient: L cone defect)1.3%0.02%
Deuteranomaly (green deficient: M cone defect)5.0%0.35%
Tritanomaly (blue deficient: S cone defect)0.0001%0.0001%

Color blindness affects a large number of individuals, with protanopia and deuteranopia being the most common types.[18] In individuals with Northern European ancestry, as many as 8 percent of men and 0.4 percent of women experience congenital color deficiency.[54]

The number affected varies among groups. Isolated communities with a restricted gene pool sometimes produce high proportions of color blindness, including the less usual types. Examples include rural Finland, Hungary, and some of the Scottish islands.[citation needed] In the United States, about 7 percent of the male population—or about 10.5 million men—and 0.4 percent of the female population either cannot distinguish red from green, or see red and green differently from how others do (Howard Hughes Medical Institute, 2006[clarification needed]). More than 95 percent of all variations in human color vision involve the red and green receptors in male eyes. It is very rare for males or females to be 'blind' to the blue end of the spectrum.[55]

Prevalence of red–green color blindness among males[56]
PopulationNumber
studied
%
Arabs (Druzes)33710.0
Aboriginal Australians4,4551.9
Belgians9,5407.4
Bosnians4,8366.2
Britons16,1806.6
Chinese1,1646.9
DR Congolese9291.7
Dutch3,1688.0
Eskimo2972.5
Fijians6080.8
French1,2438.6
Germans7,8617.7
Hutu1,0002.9
Indians (Andhra Pradesh)2927.5
Iranians16,1806.6
Japanese259,0004.0
Mexicans5712.3
Navajo5712.3
Norwegians9,0479.0
Russians1,3439.2
Scots4637.8
Swiss2,0008.0
Tibetans2415.0
Tswana4072.0
Tutsi1,0002.5
Serbs4,7507.4

History[edit]

An 1895 illustration of normal vision and various kinds of color blindness. It is not accurate[citation needed], but shows the views on this subject at the time.

The first scientific paper on the subject of color blindness, Extraordinary facts relating to the vision of colours, was published by the English chemist John Dalton in 1798[57] after the realization of his own color blindness. Because of Dalton's work, the general condition has been called daltonism, although in English this term is now used only for deuteranopia.

Society and culture[edit]

Design implications[edit]

Testing the colors of a web chart, (center), to ensure that no information is lost to the various forms of color blindness.

Color codes present particular problems for those with color deficiencies as they are often difficult or impossible for them to perceive.

Good graphic design avoids using color coding or using color contrasts alone to express information;[58] this not only helps color blind people, but also aids understanding by normally sighted people by providing them with multiple reinforcing cues.[citation needed]

Designers need to take into account that color-blindness is highly sensitive to differences in material. For example, a red–green colorblind person who is incapable of distinguishing colors on a map printed on paper may have no such difficulty when viewing the map on a computer screen or television. In addition, some color blind people find it easier to distinguish problem colors on artificial materials, such as plastic or in acrylic paints, than on natural materials, such as paper or wood. Third, for some color blind people, color can only be distinguished if there is a sufficient 'mass' of color: thin lines might appear black, while a thicker line of the same color can be perceived as having color.[citation needed]

Designers should also note that red–blue and yellow–blue color combinations are generally safe. So instead of the ever-popular 'red means bad and green means good' system, using these combinations can lead to a much higher ability to use color coding effectively. This will still cause problems for those with monochromatic color blindness, but it is still something worth considering.[59]

When the need to process visual information as rapidly as possible arises, for example in an emergency situation, the visual system may operate only in shades of gray, with the extra information load in adding color being dropped.[citation needed] This is an important possibility to consider when designing, for example, emergency brake handles or emergency phones.

Occupations[edit]

Color blindness may make it difficult or impossible for a person to engage in certain occupations. Persons with color blindness may be legally or practically barred from occupations in which color perception is an essential part of the job (e.g., mixing paint colors), or in which color perception is important for safety (e.g., operating vehicles in response to color-coded signals). This occupational safety principle originates from the Lagerlunda train crash of 1875 in Sweden. Following the crash, Professor Alarik Frithiof Holmgren, a physiologist, investigated and concluded that the color blindness of the engineer (who had died) had caused the crash. Professor Holmgren then created the first test using different-colored skeins to exclude people from jobs in the transportation industry on the basis of color blindness.[60] However, there is a claim that there is no firm evidence that color deficiency did cause the collision, or that it might have not been the sole cause.[61]

Color vision is important for occupations using telephone or computer networking cabling, as the individual wires inside the cables are color-coded using green, orange, brown, blue and white colors.[62] Electronic wiring, transformers, resistors, and capacitors are color-coded as well, using black, brown, red, orange, yellow, green, blue, violet, gray, white, silver, gold.[63]

Driving[edit]

Some countries have refused to grant driving licenses to individuals with color blindness. In Romania, there is an ongoing campaign to remove the legal restrictions that prohibit colorblind citizens from getting drivers' licenses.[64]

Ohio Bmv Eye Exam Chart

The usual justification for such restrictions is that drivers of motor vehicles must be able to recognize color-coded signals, such as traffic lights or warning lights.[59]

Piloting aircraft[edit]

While many aspects of aviation depend on color coding, only a few of them are critical enough to be interfered with by some milder types of color blindness. Some examples include color-gun signaling of aircraft that have lost radio communication, color-coded glide-path indications on runways, and the like. Some jurisdictions restrict the issuance of pilot credentials to persons who suffer from color blindness for this reason. Restrictions may be partial, allowing color-blind persons to obtain certification but with restrictions, or total, in which case color-blind persons are not permitted to obtain piloting credentials at all.[citation needed]

In the United States, the Federal Aviation Administration requires that pilots be tested for normal color vision as part of their medical clearance in order to obtain the required medical certificate, a prerequisite to obtaining a pilot's certification. If testing reveals color blindness, the applicant may be issued a license with restrictions, such as no night flying and no flying by color signals—such a restriction effectively prevents a pilot from holding certain flying occupations, such as that of an airline pilot, although commercial pilot certification is still possible, and there are a few flying occupations that do not require night flight and thus are still available to those with restrictions due to color blindness (e.g., agricultural aviation). The government allows several types of tests, including medical standard tests (e.g., the Ishihara, Dvorine, and others) and specialized tests oriented specifically to the needs of aviation. If an applicant fails the standard tests, they will receive a restriction on their medical certificate that states: 'Not valid for night flying or by color signal control'. They may apply to the FAA to take a specialized test, administered by the FAA. Typically, this test is the 'color vision light gun test'. For this test an FAA inspector will meet the pilot at an airport with an operating control tower. The color signal light gun will be shone at the pilot from the tower, and they must identify the color. If they pass they may be issued a waiver, which states that the color vision test is no longer required during medical examinations. They will then receive a new medical certificate with the restriction removed. This was once a Statement of Demonstrated Ability (SODA), but the SODA was dropped, and converted to a simple waiver (letter) early in the 2000s.[65]

Research published in 2009 carried out by the City University of London's Applied Vision Research Centre, sponsored by the UK's Civil Aviation Authority and the U.S. Federal Aviation Administration, has established a more accurate assessment of color deficiencies in pilot applicants' red/green and yellow–blue color range which could lead to a 35% reduction in the number of prospective pilots who fail to meet the minimum medical threshold.[66]

Art[edit]

Inability to distinguish color does not necessarily preclude the ability to become a celebrated artist. The 20th century expressionist painter Clifton Pugh, three-time winner of Australia's Archibald Prize, on biographical, gene inheritance and other grounds has been identified as a protanope.[67] 19th century French artist Charles Méryon became successful by concentrating on etching rather than painting after he was diagnosed as having a red–green deficiency.[68]

Rights of the color blind[edit]

Brazil[edit]

A Brazilian court ruled that people with color blindness are protected by the Inter-American Convention on the Elimination of All Forms of Discrimination against Person with Disabilities.[69][70][71]

At trial, it was decided that the carriers of color blindness have a right of access to wider knowledge, or the full enjoyment of their human condition.

United States[edit]

In the United States, under federal anti-discrimination laws such as the Americans with Disabilities Act, color vision deficiencies have not been found to constitute a disability that triggers protection from workplace discrimination.[72]

A famous traffic light on Tipperary Hill in Syracuse, New York, is upside-down due to the sentiments of its Irish American community,[73] but has been criticized due to the potential hazard it poses for color-blind persons.[74]

Research[edit]

Some tentative evidence finds that color blind people are better at penetrating certain color camouflages. Such findings may give an evolutionary reason for the high rate of red–green color blindness.[8] There is also a study suggesting that people with some types of color blindness can distinguish colors that people with normal color vision are not able to distinguish.[75] In World War II, color blind observers were used to penetrate camouflage.[76]

In September 2009, the journal Nature reported that researchers at the University of Washington and University of Florida were able to give trichromatic vision to squirrel monkeys, which normally have only dichromatic vision, using gene therapy.[77]

In 2003, a cybernetic device called eyeborg was developed to allow the wearer to hear sounds representing different colors.[78] Achromatopsic artist Neil Harbisson was the first to use such a device in early 2004; the eyeborg allowed him to start painting in color by memorizing the sound corresponding to each color. In 2012, at a TED Conference, Harbisson explained how he could now perceive colors outside the ability of human vision.[79]

See also[edit]

References[edit]

  1. ^ abcdefgGordon N (March 1998). 'Colour blindness'. Public Health. 112 (2): 81–4. doi:10.1038/sj.ph.1900446. PMID9581449.
  2. ^ abcdefghijklmnopqrstuvw'Facts About Color Blindness'. NEI. February 2015. Archived from the original on 28 July 2016. Retrieved 29 July 2016.
  3. ^Reference, Genetics Home. 'Color vision deficiency'. Genetics Home Reference. Retrieved 2019-05-06.
  4. ^'OSHA does not have requirements for normal color vision. Occupational Safety and Health Administration'. www.osha.gov. Retrieved 2019-05-06.
  5. ^Marmor MF, Lanthony P (March 2001). 'The dilemma of color deficiency and art'. Survey of Ophthalmology. 45 (5): 407–15. doi:10.1016/S0039-6257(00)00192-2. PMID11274694.
  6. ^Marmor MF (February 2016). 'Vision, eye disease, and art: 2015 Keeler Lecture'. Eye. 30 (2): 287–303. doi:10.1038/eye.2015.197. PMC4763116. PMID26563659.
  7. ^Sembulingam, K.; Sembulingam, Prema (2012). Essentials of Medical Physiology. JP Medical Ltd. p. 1002. ISBN9789350259368. Archived from the original on 2017-09-08.
  8. ^ abMorgan MJ, Adam A, Mollon JD (June 1992). 'Dichromats detect colour-camouflaged objects that are not detected by trichromats'. Proceedings. Biological Sciences. 248 (1323): 291–5. Bibcode:1992RSPSB.248.291M. doi:10.1098/rspb.1992.0074. PMID1354367.
  9. ^Choi AR, Braun JM, Papandonatos GD, Greenberg PB (November 2017). 'Occupational styrene exposure and acquired dyschromatopsia: A systematic review and meta-analysis'. American Journal of Industrial Medicine. 60 (11): 930–946. doi:10.1002/ajim.22766. PMC5652067. PMID28836685.
  10. ^Betancur-Sánchez AM, Vásquez-Trespalacios EM, Sardi-Correa C (January 2017). 'Impaired colour vision in workers exposed to organic solvents: A systematic review'. Archivos de la Sociedad Espanola de Oftalmologia. 92 (1): 12–18. doi:10.1016/j.oftal.2016.05.008. PMID27422480.
  11. ^Acquired Colour Vision DefectsArchived 2014-12-16 at the Wayback Machine. colourblindawareness.org
  12. ^MedlinePlus EncyclopediaColor blindness
  13. ^'Types of Color Deficiencies'. Konan Medical. Archived from the original on 2015-04-26. Retrieved 2014-04-26.
  14. ^Protanopia – Red–Green Color BlindnessArchived 2015-04-20 at the Wayback Machine. color-blindness.com
  15. ^Deuteranopia – Red–Green Color BlindnessArchived 2015-04-07 at the Wayback Machine. color-blindness.com
  16. ^Tovee, Martin J. (2008). An Introduction to the Visual System. Cambridge University Press. ISBN978-0-521-70964-4.
  17. ^Tritanopia – Blue–Yellow Color BlindnessArchived 2015-04-08 at the Wayback Machine. color-blindness.com
  18. ^ abcWong B (June 2011). 'Color blindness'. Nature Methods. 8 (6): 441. doi:10.1038/nmeth.1618. PMID21774112.
  19. ^Albrecht M (October 2010). 'Color blindness'. Nature Methods. 7 (10): 775, discussion 775. doi:10.1038/nmeth1010-775a. PMID20885436.
  20. ^Sharpe, L.T.; Stockman, A.; Jägle, H.; Nathans, J. (1999). 'Opsin genes, cone photopigments, color vision and color blindness'. In Gegenfurtner, K. R.; Sharpe, L. T. (eds.). Color Vision: From Genes to Perception. Cambridge University Press. ISBN978-0-521-00439-8. Archived from the original on 2016-12-27.
  21. ^American Medical Association (2003). Leikin, Jerrold B.; Lipsky, Martin S. (eds.). Complete Medical Encyclopedia (encyclopedia) (First ed.). New York, NY: Random House Reference. p. 388. ISBN978-0-8129-9100-0.
  22. ^Dick FD (March 2006). 'Solvent neurotoxicity'. Occupational and Environmental Medicine. 63 (3): 221–6, 179. doi:10.1136/oem.2005.022400. PMC2078137. PMID16497867.
  23. ^'Myambutol (Ethambutol) Drug Information: Description, User Reviews, Drug Side Effects, Interactions–Prescribing Information at RxList'. Rxlist.com. Archived from the original on 2014-07-08. Retrieved 2014-05-24.
  24. ^'Colour vision deficiency – Causes'. NHS Choices. 2012-12-14. Archived from the original on 2014-01-16. Retrieved 2014-05-24.
  25. ^ abcRoth M (13 September 2006). 'Some women may see 100,000,000 colors, thanks to their genes'. Pittsburgh Post-Gazette. Archived from the original on 8 November 2006.
  26. ^ abcDidymus, JohnThomas (Jun 19, 2012), 'Scientists find woman who sees 99 million more colors than others', Digital Journal, archived from the original on 2016-02-08
  27. ^ abcJordan G, Deeb SS, Bosten JM, Mollon JD (July 2010). 'The dimensionality of color vision in carriers of anomalous trichromacy'. Journal of Vision. 10 (8): 12. doi:10.1167/10.8.12. PMID20884587.
  28. ^Gordon N (March 1998). 'Colour blindness'. Public Health. 112 (2): 81–4. doi:10.1038/sj.ph.1900446. PMID9581449.
  29. ^Kinnear PR, Sahraie A (December 2002). 'New Farnsworth–Munsell 100 hue test norms of normal observers for each year of age 5-22 and for age decades 30-70'. The British Journal of Ophthalmology. 86 (12): 1408–11. doi:10.1136/bjo.86.12.1408. PMC1771429. PMID12446376.
  30. ^Cole BL, Lian KY, Lakkis C (March 2006). 'The new Richmond HRR pseudoisochromatic test for colour vision is better than the Ishihara test'. Clinical & Experimental Optometry. 89 (2): 73–80. doi:10.1111/j.1444-0938.2006.00015.x. PMID16494609.
  31. ^Toufeeq A (October 2004). 'Specifying colours for colour vision testing using computer graphics'. Eye. 18 (10): 1001–5. doi:10.1038/sj.eye.6701378. PMID15192692.
  32. ^Spring KR, Parry-Hill MJ, Fellers TJ, Davidson MW. 'Human Vision and Color Perception'. Florida State University. Archived from the original on 2007-08-27. Retrieved 2007-04-05.
  33. ^Hoffman, Paul S. 'Accommodating Color Blindness'(PDF). Archived from the original(PDF) on 15 May 2008. Retrieved 2009-07-01.
  34. ^Neitz, Maureen E. 'Severity of Colorblindness Varies'. Medical College of Wisconsin. Archived from the original on 5 February 2007. Retrieved 2007-04-05.
  35. ^Jones SA, Shim SH, He J, Zhuang X (June 2011). 'Fast, three-dimensional super-resolution imaging of live cells'. Nature Methods. 8 (6): 499–508. doi:10.1038/nmeth.1605. PMC3137767. PMID21552254.
  36. ^Simunovic MP (May 2010). 'Colour vision deficiency'. Eye. 24 (5): 747–55. doi:10.1038/eye.2009.251. PMID19927164.
  37. ^Neitz J, Neitz M (April 2011). 'The genetics of normal and defective color vision'. Vision Research. 51 (7): 633–51. doi:10.1016/j.visres.2010.12.002. PMC3075382. PMID21167193.
  38. ^ abcHarrison G, Tanner J, Pilbeam D, Baker P (1988). Human Biology. Oxford: Oxford University Press. pp. 183–187, 287–290. ISBN978-0-19-854144-8.
  39. ^Jameson KA, Highnote SM, Wasserman LM (June 2001). 'Richer color experience in observers with multiple photopigment opsin genes'(PDF). Psychonomic Bulletin & Review. 8 (2): 244–61. doi:10.3758/BF03196159. PMID11495112. Archived from the original(PDF) on 14 February 2012. Retrieved 25 March 2017.
  40. ^Almutairi, Nawaf; Kundart, James; Muthuramalingam, Naganathan; Hayes, John; Citek, Karl (24 July 2017). 'Assessment of Enchroma Filter for Correcting Color Vision Deficiency'. Pacific University College of Optometry. 21. Retrieved 5 January 2019.
  41. ^MacAdam, David L.; Judd, Deane B., eds. (1979). Contributions to color science. NBS. p. 584.
  42. ^ abcKalloniatis M, Luu C (July 9, 2007). 'The Perception of Color'. In Kolb H, Fernandez E, Nelson R (eds.). Webvision: The Organization of the Retina and Visual System. University of Utah Health Sciences Center. PMID21413396. Archived from the original on May 9, 2018.
  43. ^Goldstein EB (2007). Sensation and perception (7th ed.). Wadsworth: Thomson. p. 152. ISBN978-0-534-55810-9.
  44. ^'Disease-causing Mutations and protein structure'. UCL Biochemistry BSM Group. Retrieved 2007-04-02.[permanent dead link]
  45. ^'Types of Colour Blindness'. Colour Blind Awareness. Archived from the original on 2014-05-29.
  46. ^ abBlom, Jan Dirk (2009). A Dictionary of Hallucinations. Springer. p. 4. ISBN978-1-4419-1222-0. Archived from the original on 2016-12-27.
  47. ^Weiss AH, Biersdorf WR (1989). 'Blue cone monochromatism'. Journal of Pediatric Ophthalmology and Strabismus. 26 (5): 218–23. PMID2795409.
  48. ^Color Vision DeficiencyArchived 2015-04-16 at the Wayback Machine. American Optometric Association
  49. ^Siegel IM (1981). 'The X-Chrom lens. On seeing red'. Survey of Ophthalmology. 25 (5): 312–24. doi:10.1016/S0039-6257(81)80001-X. PMID6971497.
  50. ^Zeltzer HI (July 1979). 'Use of modified X-Chrom for relief of light dazzlement and color blindness of a rod monochromat'. Journal of the American Optometric Association. 50 (7): 813–8. PMID315420.
  51. ^An X-Chrom manualArchived 2015-04-12 at the Wayback Machine. Artoptical.com. Retrieved on 2016-12-10.
  52. ^Zhou L. 'A Scientist Accidentally Developed Sunglasses That Could Correct Color Blindness'. Smithsonian. Retrieved 6 January 2018.
  53. ^'Causes and Incidence of Colorblindness'. Causes of Color. Archived from the original on 18 November 2013. Retrieved 27 February 2014.[unreliable source?]
  54. ^Chan X, Goh S, Tan N (2014). 'Subjects with colour vision deficiency in the community: what do primary care physicians need to know?'. Asia Pacific Family Medicine. 13 (1): 10. doi:10.1186/s12930-014-0010-3.
  55. ^Ananya, Mandal (2009-11-23). 'Color Blindness Prevalence'. Health. Archived from the original on 8 September 2013. Retrieved 27 February 2014.
  56. ^Harrison, G.A. et al. (1977): Human Biology, Oxford University Press, Oxford, ISBN0-19-857164-X.
  57. ^Dalton J (1798). 'Extraordinary facts relating to the vision of colours: with observations'. Memoirs of the Literary and Philosophical Society of Manchester. 5: 28–45. OCLC9879327.
  58. ^Crow KL (2008). 'Four Types of Disabilities: Their Impact on Online Learning'. TechTrends. 52 (1): 51–5. doi:10.1007/s11528-008-0112-6.
  59. ^ abHabibzadeh P (September 2016). 'Our red–green world'. Australian Health Review. 40 (4): 474. doi:10.1071/ah15161. PMID26476553.
  60. ^Vingrys AJ, Cole BL (1986). 'Origins of colour vision standards within the transport industry'. Ophthalmic & Physiological Optics. 6 (4): 369–75. doi:10.1111/j.1475-1313.1986.tb01155.x. PMID3306566.
  61. ^Mollon JD, Cavonius LR (2012). 'The Lagerlunda collision and the introduction of color vision testing'. Survey of Ophthalmology. 57 (2): 178–94. doi:10.1016/j.survophthal.2011.10.003. PMID22301271.
  62. ^Meyers, Michael (2002). All in One A+ Certification Exam Guide (4th ed.). Berkeley, California: McGraw-Hill/Osborne. ISBN978-0-07-222274-6.[page needed]
  63. ^Grob, Bernard (2001). Basic Electronics. Columbus, Ohio: Glencoe/McGraw-Hill. ISBN978-0-02-802253-6.[page needed]
  64. ^'Petition to European Union on Colorblind's condition in Romania'. 2007-08-15. Archived from the original on 2007-10-13. Retrieved 2007-08-21.[self-published source?]
  65. ^'Aerospace Medical Dispositions — Color vision'. Archived from the original on 2009-05-12. Retrieved 2009-04-11.
  66. ^Warburton, Simon (29 May 2009). 'Colour-blindness research could clear more pilots to fly: UK CAA'. Air transport. Reed Business Information. Archived from the original on 2 June 2009. Retrieved 29 October 2009.
  67. ^Cole BL, Harris RW (September 2009). 'Colour blindness does not preclude fame as an artist: celebrated Australian artist Clifton Pugh was a protanope'. Clinical & Experimental Optometry. 92 (5): 421–8. doi:10.1111/j.1444-0938.2009.00384.x. PMID19515095.
  68. ^Anon. 'Charles Meryon'. Art Encyclopedia. The Concise Grove Dictionary of Art. Oxford University Press. Archived from the original on 25 November 2010. Retrieved 7 January 2010.
  69. ^'Full text of the decision of the court – in Portuguese language'. Archived from the original on 2012-07-14. Retrieved 2012-03-09.
  70. ^'Decree issued by president of a republic ratifying Legislative Decree No. 198, of june 13, which approved the Inter-American Convention AG/RES. 1608 – in Portuguese language'. Archived from the original on 2012-03-25. Retrieved 2012-03-09.
  71. ^'Inter-American Convention on the Elimination of All Forms of Discrimination against Person with Disabilities'. Archived from the original on 2013-04-16. Retrieved 2012-03-09.
  72. ^Larson A (22 May 2016). 'Is Color-Blindness a Disability for Employment Under the ADA'. ExpertLaw. ExpertLaw.com. Archived from the original on 15 August 2017. Retrieved 15 August 2017.
  73. ^'New documentary uncovers the Irish links to America's Tipperary Hill'. TheJournal.ie. 6 November 2016. Archived from the original on 15 August 2017. Retrieved 15 August 2017.
  74. ^Zhang, Sarah. 'The Story Behind Syracuse's Upside-Down Traffic Light'. Gizmodo. Archived from the original on 2014-09-16.
  75. ^Bosten JM, Robinson JD, Jordan G, Mollon JD (December 2005). 'Multidimensional scaling reveals a color dimension unique to 'color-deficient' observers'. Current Biology. 15 (23): R950–2. doi:10.1016/j.cub.2005.11.031. PMID16332521.
  76. ^'Colour blindness not all it seems'. BBC News. 6 December 2015. Archived from the original on 23 June 2016. Retrieved 21 June 2016.
  77. ^Dolgin E (2009). 'Colour blindness corrected by gene therapy'. Nature. doi:10.1038/news.2009.921.
  78. ^Alfredo M. Ronchi: Eculture: Cultural Content in the Digital Age. Springer (New York, 2009). p. 319 ISBN978-3-540-75273-8
  79. ^'I listen to color'Archived 2012-08-12 at the Wayback Machine, Neil Harbisson at TED Global, 27 June 2012.

Further reading[edit]

  • Kaiser PK, Boynton RM (1996). Human color vision. Washington, DC: Optical Society of America. ISBN978-1-55752-461-4. OCLC472932250.
  • McIntyre D (2002). Colour blindness: causes and effects. Chester: Dalton Publishing. ISBN978-0-9541886-0-3. OCLC49204679.
  • Rubin ML, Cassin B, Solomon S (1984). Dictionary of eye terminology. Gainesville, Fla: Triad Pub. Co. ISBN978-0-937404-07-2. OCLC10375427.
  • Shevell SK (2003). The science of color. Amsterdam: Elsevier. ISBN978-0-444-51251-2. OCLC52271315.
  • Hilbert D, Byrne A (1997). Readings on color. Cambridge, Mass: MIT Press. ISBN978-0-262-52231-1. OCLC35762680.
  • Stiles WS, Wyszecki G (2000). Color science: concepts and methods, quantitative data and formulae. Chichester: John Wiley & Sons. ISBN978-0-471-39918-6. OCLC799532137.
  • Kuchenbecker J, Broschmann D (2014). Plates for color vision testing. New York: Thieme. ISBN978-3-13-175481-3.

Ohio Drivers Eye Exam Chart Test

External links[edit]

Wikimedia Commons has media related to Color blindness.

Ohio Drivers Eye Exam Chart Online

Wikisource has original text related to this article:
  • Color blindness at Curlie
Classification
  • ICD-10: H53.5
  • ICD-9-CM: 368.5
  • MeSH: D003117
  • DiseasesDB: 2999
External resources
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